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dc.contributor.authorTuğlular, Serhan
dc.contributor.authorÖzen-AI Ahbad, Yeşim
dc.contributor.authorKantarcı, Gülçin
dc.contributor.authorKoç, Mehmet
dc.contributor.authorAvşar, Erol
dc.contributor.authorÖzener, Çetin
dc.contributor.authorAkoğlu, Emel
dc.date.accessioned2016-06-03T10:32:02Z
dc.date.available2016-06-03T10:32:02Z
dc.date.issued2003
dc.identifier.urihttp://hdl.handle.net/11424/4595
dc.description.abstractWe report an 18 year old male patient with a known diagnosis of BRIC who presented with acute renal failure secondary to hyperbilirubinemia In three successive episodes. Renal replacement therapy was required in all three episodes but his renal function recovered to baseline creatinine on discharge. Proposed pathophysiology of ARF in the setting of hyperbilirubinemia includes direct tubulotoxlcity and sequestration of pigment casts within the tubular lumen causing tubular obstruction aggravated by dehydration. We emphasize the importance of vigorous hydration to be started with the impending attack to prevent progression to ARF.en_US
dc.language.isoengen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectAcute Renal Failure, BRICen_US
dc.titleBenıgn recurrent ıntrahepatıc cholestasıs (brıc): Is ıt really as benıgn as antıcıpated?en_US
dc.typearticleen_US
dc.contributor.authorIDTR172362en_US
dc.contributor.authorIDTR219206en_US
dc.contributor.authorIDTR4220en_US
dc.contributor.authorIDTR206603en_US
dc.relation.journalMarmara Medical Journalen_US
dc.identifier.volume16en_US
dc.identifier.issue2en_US
dc.identifier.startpage121en_US
dc.identifier.endpage123en_US


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